Medical Care |

Medical Care

##SEVER##

/h/hlr.nu1.html

 

Hlr.nu

Part 17: First Aid
2010 American Heart Association and American Red Cross Guidelines for
First Aid
David Markenson, Co-Chair*; Jeffrey D. Ferguson, Co-Chair*; Leon Chameides; Pascal Cassan; Kin-Lai Chung; Jonathan Epstein; Louis Gonzales; Rita Ann Herrington; Jeffrey L. Pellegrino; Norda Ratcliff; Adam Singer Modern, organized first aid evolved from military experi- Red Cross (Red Cross) cofounded the National First Aid ences when surgeons taught soldiers how to splint and Science Advisory Board to review and evaluate the scien- bandage battlefield wounds. Two British officers, Peter Shep- tific literature on first aid in preparation for the 2005 herd and Francis Duncan, are said to have been the first to American Heart Association (AHA) and American Red expand the concept to civilians and to develop the first Cross Guidelines for First Aid.1 In preparation for the 2010 curriculum in first aid.4 Organized training in civilian first aid evidence evaluation process, the National First Aid Advi- began in the United States in 1903 when Clara Barton, sory Board was expanded to become the International First president of the Red Cross, formed a committee to establish Aid Science Advisory Board with the addition of repre- instruction in first aid among the nation's industrial workers, sentatives from a number of international first aid organi- where, under dangerous conditions, accidents and deaths zations (see Table). The goal of the board is to reduce were all too frequent.
morbidity and mortality due to emergency events bymaking treatment recommendations based on an analysis The Evidence Evaluation Process
of the scientific evidence that answers the following The International First Aid Science Advisory Board first identified 38 questions in first aid practice that either were not raised in previous evidence evaluations or were in need of In which emergency conditions can morbidity or mortality updating. Two or more board members volunteered to review be reduced by the intervention of a first aid provider? the scientific literature independently and develop an How strong is the scientific evidence that interventions evidence-based review worksheet summarizing the literature performed by a first aid provider are safe, effective, and relevant to each question (see Part 2: "Evidence Evaluation and Management of Potential or Perceived Conflicts of A critical review of the scientific literature by members of Interest"). After each worksheet was presented to, and re- the International First Aid Science Advisory Board is sum- viewed by, the full board, a summary draft of the scientific marized in the 2010 International Consensus on First Aid evidence and a treatment recommendation were crafted. The Science With Treatment Recommendations (ILCOR 2010 evidence-based review for each question was presented and CPR Consensus), from which these guidelines are derived.2 discussed a second time at a subsequent board meeting. All That critical review evaluates the literature and identifies first aid worksheets, co-copyrighted by the American Heart knowledge gaps that might be filled through future scientific Association and the American Red Cross, can be viewed through hyperlinks in the 2010 American Heart Associationand American Red Cross International Consensus on First Aid Science with Treatment Recommendations.2 Each ques- The history of first aid can be traced to the dawn of organized tion, evidence-based review, draft summary of science, and human societies. For example, Native American Sioux med- draft treatment recommendation was presented, discussed, icine men of the Bear Society were noted for treating battle and debated on 2 separate occasions until a consensus was injuries, fixing fractures, controlling bleeding, removing ar- reached. These guidelines are based on the scientific consen- rows, and using a sharp flint to cut around wounds and sus findings reported in the 2010 International Consensus on First Aid Science with Treatment Recommendations.2 The American Heart Association and the American Red Cross request that this document be cited as follows: Markenson D, Ferguson JD, Chameides L, Cassan P, Chung K-L, Epstein J, Gonzales L, Herrington RA, Pellegrino JL, Ratcliff N, Singer A. Part 17: first aid: 2010 American Heart Associationand American Red Cross Guidelines for First Aid. Circulation. 2010;122(suppl 3):S934 –S946.
*Co-chairs and equal first co-authors.
(Circulation. 2010;122[suppl ]:S934 –S946.)
2010 American Heart Association, Inc., and American Red Cross.
Circulation is available at http://circ.ahajournals.org
Markenson et al
Part 17: First Aid
International First Aid Science Advisory Board
sound and based on scientific evidence or, in the absence of such evidence, on expert consensus. Administration offirst aid must not delay activation of the emergency American Academy of Pediatrics medical services (EMS) system or other medical assistance American Burn Association when required. We strongly believe that education in first American College of Emergency Physicians aid should be universal: everyone can learn first aid and American College of Occupational and Environmental Medicine everyone should.
American College of Surgeons The scope of first aid is not purely scientific; it is American Heart Association influenced by both training and regulatory issues. The defi- American Pediatric Surgical Association nition of scope is therefore variable, and should be defined American Red Cross according to circumstances, need, and regulatoryrequirements.
American Red Cross Advisory Council on First Aid, Aquatics, Safety andPreparedness (ACFASP) American Safety & Health Institute (ASHI) Calling for Help
Austrian Red Cross A first aid provider must be able to recognize when help is Canadian Red Cross needed and how to get it. First aid providers should learn Divers Alert Network how and when to access the EMS system, how to activatethe on-site emergency response plan (ERP), and how to European Reference Center on First Aid Education contact the Poison Control Center (see "Poison Emergen- Egyptian Red Crescent cies" below).
Grenada Red Cross Positioning the Victim
Hong Kong Red Cross As a general rule a victim should not be moved, especially if Hungarian Red Cross you suspect, from the victim's position or the nature of the International Federation of Red Cross and Red Crescent Societies injury, that the victim may have a spinal injury (see "Spine Medic First Aid International Stabilization" below). There are times, however, when the National Association of EMS Educators victim should be moved: National Association of EMS Physicians If the area is unsafe for the rescuer or the victim, move the National Athletic Trainers' Association victim to a safe location if it is safe to do so.
National Safety Council If the victim is face down and is unresponsive, turn the Norwegian Red Cross victim face up.
Occupational Safety and Health Administration If the victim has difficulty breathing because of copious Red Cross Society of China secretions or vomiting, or if you are alone and have to leave Resuscitation Council of Asia an unresponsive victim to get help, place the victim in a St. John Ambulance, UK modified High Arm IN Endangered Spine (HAINES)
recovery position:9,10 Extend one of the victim's arms
above the head and roll the body to the side so the victim's
Previous reports5–8 have noted the paucity of scientific head rests on the extended arm. Bend both legs to stabilize evidence supporting many interventions in prehospital emer- the victim (Class IIb, LOE C).
gency care. In reviewing the medical literature, members of If a victim shows evidence of shock, have the victim lie the International First Aid Science Advisory Board once supine. If there is no evidence of trauma or injury, raise the again found a paucity of evidence to guide first aid interven- feet about 6 to 12 inches (about 30° to 45°) (Class IIb, LOE tions. Very little research is being conducted in first aid, and C). Do not raise the feet if the movement or the position many of the following recommendations are extrapolated causes the victim any pain.
from the experience of healthcare professionals. It is impor-tant to recognize the limitations of the evidence that supports The evidence for a benefit to raising the feet is extrapolated many of these guidelines so that research can be undertaken from leg raising studies on volume expansion; there are no and future guidelines can be based on a larger body of studies on the effect of leg raising as a first aid maneuver for shock. The results of the volume expansion studies arecontradictory with some showing an increase in cardiac Definition of First Aid
output,11–13 while others show no change in cardiac output ormean arterial pressure14–18 with leg raising.
We define first aid as the assessments and interventionsthat can be performed by a bystander (or by the victim)with minimal or no medical equipment. A first aid provider is defined as someone with formal training in first aid, There is insufficient evidence to recommend routine use of emergency care, or medicine who provides first aid. First supplementary oxygen by a first aid provider for victims aid assessments and interventions should be medically complaining of chest discomfort19,20 or shortness of breath21 November 2, 2010
(Class IIb, LOE C). Supplementary oxygen administration epinephrine may be given if symptoms of anaphylaxis may be beneficial as part of first aid for divers with a persist (Class IIb, LOE C).
decompression injury (Class IIb, LOE C22).
The general principles of first aid management of seizuresare to Ensure an open airway.
The incidence of acute asthma is increasing, especially in Prevent injury.
urban populations.23 Many victims with asthma take a pre-scribed bronchodilator medication and can self-administer Do not restrain the victim during a seizure. Do not try to it.24–26 First aid providers are not expected to make a open the victim's mouth or try to place any object between diagnosis of asthma, but they may assist the victim in using the victim's teeth or in the mouth. Restraining the victim the victim's prescribed bronchodilator medication (Class IIa, may cause musculoskeletal or soft-tissue injury. Placing an LOE B) under the following conditions: object in the victim's mouth may cause dental damage or The victim states that he or she is having an asthma attack aspiration (Class IIa, LOE C). It is not unusual for the or symptoms associated with a previously diagnosed victim to be unresponsive or confused for a short time after breathing disorder, and the victim has the prescribed medications or inhaler in his or her possession.
The victim identifies the medication and is unable to administer it without assistance.24 Because it is very difficult, even for the healthcare profes-sional, to differentiate chest discomfort of cardiac origin from First aid providers should become familiar with inhalers so other chest discomfort, the first aid provider should assume that they can assist a victim with an acute asthma attack in that chest discomfort is cardiac until proven otherwise.
using the inhaler.
Cardiac chest discomfort is often described as "crushing" or"pressing" and is often accompanied by shortness of breath or perspiration. But cardiac chest discomfort may not have these Allergies are relatively common, but only a small proportion classical characteristics, especially in women. Call EMS of people with allergies develop anaphylactic reactions. An immediately for anyone with chest discomfort. Do not delay anaphylactic reaction is a progressive series of signs and and do not try to transport the patient to a healthcare facility symptoms characterized by swelling, breathing difficulty, an itching rash, and eventually shock, which, if left While waiting for EMS to arrive, the first aid provider may untreated, may lead to death. Some of these signs and encourage the victim to chew 1 adult (not enteric coated) or symptoms can also be present in other conditions, and first 2 low-dose "baby" aspirin if the patient has no allergy to aid rescuers should not be expected to make a diagnosis of aspirin or other contraindication to aspirin, such as evidence of a stroke or recent bleeding (Class IIa, LOE A).44–46 Older patients who suffer from anaphylactic reactions know their signs and symptoms and many carry a lifesavingepinephrine auto-injector. With proper training, parents can be taught to correctly use an auto-injector to administerepinephrine to their allergic children.31 All too often, how- ever, neither the victim nor family members know how to Control of bleeding is a basic skill of first aid and one of the correctly use an auto-injector.32–34 First aid providers should few actions with which a first aid provider can critically be familiar with the epinephrine auto-injector so that they can influence outcome.
help a victim with an anaphylactic reaction to self-administerit. First aid providers should also know how to administer the auto-injector if the victim is unable to do so, provided that the Bleeding is best controlled by applying pressure until bleed- medication has been prescribed by a physician and state law ing stops47–53 or EMS rescuers arrive (Class I, LOE A). The permits it (Class IIb, LOE B).
amount of pressure applied and the time the pressure is held In retrospective studies35–37 18% to 35% of patients are the most important factors affecting successful control of having signs of anaphylaxis required a second dose of bleeding. The pressure must be firm, and it must be main- epinephrine if symptoms persisted or progressed after the tained for a long time. Methods of applying pressure include first dose. Because of the difficulty in making a diagnosisof anaphylaxis27–30,38,39 and the potential harm from epi- Manual pressure on gauze or other cloth placed over the nephrine if the diagnosis is incorrect,40 – 43 first aid provid- bleeding source. If bleeding continues, do not remove the ers are advised to seek medical assistance if symptoms gauze; add more gauze on top and apply more pressure.
persist, rather than routinely administering a second dose If it is not possible to provide continuous manual pressure, of epinephrine. In unusual circumstances, when advanced wrap an elastic bandage firmly over gauze to hold it in medical assistance is not available, a second dose of place with pressure.54–57 Markenson et al
Part 17: First Aid
Although tourniquets have been shown to control bleedingeffectively on the battlefield58–60 and during surgery and have been used by paramedics in a civilian setting without com- Cool thermal burns with cold (15° to 25°C) tap water as soon plications,61 there are no studies on controlling bleeding with as possible and continue cooling at least until pain is relieved first aid provider use of a tourniquet. Potential dangers of (Class I, LOE B).86–93 Cooling reduces pain, edema, and prolonged tourniquet application include temporary62 or per- depth of injury. It speeds healing and may reduce the need for manent63 injury to the underlying nerves and muscles,64 and excision and grafting of deep burns. Don't apply ice directly systemic complications resulting from limb ischemia,65 in- to a burn; it can produce tissue ischemia (Class III, LOE B).
cluding acidemia, hyperkalemia, arrhythmias, shock, and Prolonged cold exposure to small burns, and even brief death. Complications are related to tourniquet pressure66 and exposure if the burn is large, can cause further local tissue duration of occlusion,67 but there is insufficient evidence to injury93–95 and hypothermia.
determine a minimal critical time beyond which irreversiblecomplications may occur. Because of the potential adverse effects of tourniquets and difficulty in their proper applica- Loosely cover burn blisters with a sterile dressing but leave tion, use of a tourniquet to control bleeding of the extremities blisters intact because this improves healing and reduces pain is indicated only if direct pressure is not effective or possible (Class IIa, LOE B).96–99 (Class IIb, LOE B). Specifically designed tourniquets appearto be better than ones that are improvised,60,68–71 but tourni- quets should only be used with proper training (Class IIa, The severity of electric injuries can vary widely, from an LOE B). If a tourniquet is used, make sure that you note the unpleasant tingling sensation caused by low-intensity cur- time it was applied and communicate that time to EMS rent to thermal burns, cardiopulmonary arrest, and death.
Thermal burns may result from burning clothing that is incontact with the skin or from electric current traversing a Pressure Points and Elevation
portion of the body. When current traverses the body, Elevation and use of pressure points are not recommended to thermal burns may be present at the entry and exit points control bleeding (Class III, LOE C). This new recommenda- and along its internal pathway. Cardiopulmonary arrest is tion is made because there is evidence that other ways of the primary cause of immediate death from electrocu- controlling bleeding are more effective. The hemostatic effect tion.100 Cardiac arrhythmias, including ventricular fibrilla- of elevation has not been studied. No effect on distal pulses tion, ventricular asystole, and ventricular tachycardia that was found in volunteers when pressure points were used.72 progresses to ventricular fibrillation, may result from Most important, these unproven procedures may compromise exposure to low- or high-voltage current.101 Respiratory the proven intervention of direct pressure, so they could be arrest may result from electric injury to the respiratory center in the brain or from tetanic contractions or paralysisof respiratory muscles.
Do not place yourself in danger by touching an electro- Among the large number of commercially available hemo- cuted victim while the power is on (Class III, LOE C).
static agents, some have been shown to be effective.73–76 Turn off the power at its source; at home the switch is However, their routine use in first aid cannot be recom- usually near the fuse box. In case of high-voltage electro- mended at this time because of significant variation in cutions caused by fallen power lines, immediately notify effectiveness by different agents and their potential for the appropriate authorities (eg, 911 or fire department). All adverse effects, including tissue destruction with induction of materials conduct electricity if the voltage is high enough, a proembolic state and potential thermal injury (Class IIb, so do not enter the area around the victim or try to remove wires or other materials with any object, including awooden one, until the power has been turned off by Wounds and Abrasions
Superficial wounds and abrasions should be thoroughly Once the power is off, assess the victim, who may need irrigated with a large volume of warm or room temperature CPR, defibrillation, and treatment for shock and thermal potable water with or without soap77–82 until there is no burns. All victims of electric shock require medical assess- foreign matter in the wound (Class I, LOE A). Cold water ment because the extent of injury may not be apparent.
appears to be as effective as warm water, but it is not ascomfortable. If running water is unavailable, use any source of clean water. Wounds heal better with less infection if they There is approximately a 2% risk of injury to the cervical are covered with an antibiotic ointment or cream and a clean spine after blunt trauma that is serious enough to require occlusive dressing (Class IIa, LOE A).83–85 Apply antibiotic spinal imaging in an emergency department,102,103 and this ointment or cream only if the wound is an abrasion or a risk is tripled in patients with craniofacial injury.104 Most superficial injury and only if the victim has no known victims with spinal injuries are males between the ages of allergies to the antibiotic.
10 and 30 years. Motor vehicles cause approximately half November 2, 2010
of all spinal injuries; many of the remainder are caused by thin towel, between the cold container and the skin (Class IIb, falls (especially from a height or diving), sports, and LOE C126,128).
It is not clear whether a compression bandage is helpful for If the cervical spine is injured, the spinal cord may be a joint injury. Heat application to a contusion or injured joint unprotected, and further injury (secondary spinal cord is not as good a first aid measure as cold application.115 injury) could result from stresses to the cord that occurwhen the victim is manipulated or moved. This could result in permanent neurological damage including quad- Assume that any injury to an extremity includes a bone riplegia.106,107 Only one controlled but underpowered study fracture. Cover open wounds with a dressing. Do not move with some methodological problems108 has examined this or try to straighten an injured extremity (Class III, LOE C).
question. In the study, the group of injured victims with There is no evidence that straightening an angulated spinal immobilization by emergency medical technicians suspected long bone fracture shortens healing time or using equipment failed to show any neurological benefit reduces pain prior to permanent fixation. Expert opinion compared with a group of injured victims without spinal suggests that splinting may reduce pain130 and prevent further injury. So, if you are far from definitive health care, Because of the dire consequences if secondary injury stabilize the extremity with a splint in the position found does occur, maintain spinal motion restriction by manually (Class IIa, LOE C). If a splint is used, it should be padded stabilizing the head so that the motion of head, neck, and to cushion the injury. If an injured extremity is blue or spine is minimized (Class IIb, LOE C). First aid providers extremely pale, activate EMS immediately because this should not use immobilization devices because their ben- could be a medical emergency. A victim with an injured efit in first aid has not been proven and they may be lower extremity should not bear weight until advised to do harmful (Class III, LOE C). Immobilization devices may so by a medical professional.
be needed in special circumstances when immediate extri-cation (eg, rescue of drowning victim) is required, but first Human and Animal Bites
aid providers should not use these devices unless they have Irrigate human and animal bites with copious amounts of been properly trained in their use.
water (Class I, LOE B). This irrigation has been shown to First aid rescuers cannot conclusively identify a victim prevent rabies from animal bites131,132 and bacterial with a spinal injury, but they should suspect spinal injury if an injured victim has any of the following risk factors(these have been modified slightly from the 2005 Ameri- can Heart Association and American Red Cross First Aid Do not apply suction as first aid for snakebites (Class III, LOE C). Suction does remove some venom, but the amountis very small.134 Suction has no clinical benefit135 and it may aggravate the injury.136–138 Driver, passenger, or pedestrian, in a motor vehicle, mo- Applying a pressure immobilization bandage with a pres- torized cycle, or bicycle crash sure between 40 and 70 mm Hg in the upper extremity and Fall from a greater than standing height between 55 and 70 mm Hg in the lower extremity around the Tingling in the extremities entire length of the bitten extremity is a reasonable way to Pain or tenderness in the neck or back slow the dissemination of venom by slowing lymph flow Sensory deficit or muscle weakness involving the torso or (Class IIa, LOE C139,140). For practical purposes pressure is upper extremities sufficient if the bandage is comfortably tight and snug but Not fully alert or is intoxicated allows a finger to be slipped under it. Initially it was theorized Other painful injuries, especially of the head and neck that slowing lymphatic flow by external pressure would only Children 2 years of age or older with evidence of head or benefit victims bitten by snakes producing neurotoxic venom,140 but the effectiveness of pressure immobilizationhas also been demonstrated for bites by non-neurotoxic American snakes in an animal model.141 This treatmentrequires further study to prove its efficacy in humans. The Sprains and Strains
challenge is to find a way to teach the application of the Soft-tissue injuries include joint sprains and muscle contu- correct snugness of the bandage because inadequate pressure sions. Cold application decreases hemorrhage, edema, pain, is ineffective and too much pressure may cause local tissue and disability,115–120 and it is reasonable to apply cold to a damage. It has also been demonstrated that, once learned, soft-tissue injury. Cooling is best accomplished with a plastic retention of the skill of proper pressure and immobilization bag or damp cloth filled with a mixture of ice and water; the application is poor.142,143 mixture is better than ice alone.121–123 Refreezable gel packsdo not cool as effectively as an ice-water mixture.124,125 To prevent cold injury, limit each application of cold to periods This section is new to the First Aid Guidelines. First aid ⱕ20 minutes.126–128 If that length of time is uncomfortable, for jellyfish stings consists of two important actions: limit application to 10 minutes.129 Place a barrier, such as a preventing further nematocyst discharge and pain relief.
Markenson et al
Part 17: First Aid
To inactivate venom load and prevent further envenoma- clude placing the victim near a heat source and placing tion, jellyfish stings should be liberally washed with containers of warm, but not hot, water in contact with the vinegar (4% to 6% acetic acid solution) as soon as possible for at least 30 seconds (Class IIa, LOE B). The inactivationof venom has been demonstrated for Olindias sambaquien- sis144 and for Physalia physalis (Portuguese man-of- Frostbite usually affects an exposed part of the body such as war).145 If vinegar is not available, a baking soda slurry the extremities and nose. In case of frostbite, remove wet may be used instead.145 clothing and dry and cover the victim to prevent hypothermia.
For the treatment of pain, after the nematocysts are Transport the victim to an advanced medical facility as removed or deactivated, jellyfish stings should be treated with rapidly as possible. Do not try to rewarm the frostbite if there hot-water immersion when possible (Class IIa, LOE B). The is any chance that it might refreeze161,162 or if you are close to victim should be instructed to take a hot shower or immerse a medical facility (Class III, LOE C).
the affected part in hot water (temperature as hot as tolerated, Minor or superficial frostbite (frostnip) can be treated with or 45°C if there is the capability to regulate temperature), as simple, rapid rewarming using skin-to-skin contact such as a soon as possible, for at least 20 minutes or for as long as pain persists.146–149 If hot water is not available, dry hot packs or, Severe or deep frostbite should be rewarmed within 24 as a second choice, dry cold packs may be helpful in hours of injury and this is best accomplished by immersing decreasing pain but these are not as effective as hot water the frostbitten part in warm (37° to 40°C or approximately (Class IIb, LOE B146,150,151). Topical application of aluminum body temperature) water for 20 to 30 minutes (Class IIb, LOE sulfate or meat tenderizer, commercially available aerosol C161–170). Chemical warmers should not be placed directly on products, fresh water wash, and papain, an enzyme derived frostbitten tissue because they can reach temperatures that from papaya used as a local medicine, are even less effective can cause burns (Class III, LOE C171). Following rewarming, in relieving pain (Class IIb, LOE B147,152).
efforts should be made to protect frostbitten parts from Pressure immobilization bandages are not recommended refreezing and to quickly evacuate the patient for further care.
for the treatment of jellyfish stings because animal stud- The effectiveness of ibuprofen or other nonsteroidal antiin- ies153,154 show that pressure with an immobilization bandage flammatory drugs (NSAIDs) in frostbite has not been well causes further release of venom, even from already fired established in human studies.170,172–175 nematocysts (Class III, LOE C).
Heat-induced symptoms, often precipitated by vigorous ex- Traumatic dental injuries are common. The first aid for an ercise, may include heat cramps, heat exhaustion, and heat avulsed tooth is as follows: Heat cramps are painful involuntary muscle spasms that Clean bleeding wound(s) with saline solution or tap water.
most often affect the calves, arms, abdominal muscles, and Stop bleeding by applying pressure with gauze or cotton.
back. First aid includes rest, cooling off, and drinking an Handle the tooth by the crown, not the root (ie, do not electrolyte-carbohydrate mixture, such as juice, milk, or a handle the part that was beneath the gum).
Place the tooth in milk, or clean water if milk is not commercial electrolyte-carbohydrate drink.176–185 Stretching, icing, and massaging the painful muscles may be helpful.
Contact the patient's dentist or take the tooth and victim to Exercise should not be resumed until all symptoms have an emergency care center as quickly as possible (Class IIa, Heat exhaustion is caused by a combination of exercise- induced heat and fluid and electrolyte loss as sweat. Signs andsymptoms may start suddenly and include: nausea, dizziness, muscle cramps, feeling faint, headache, fatigue, and heavysweating. Heat exhaustion is a serious condition because it can rapidly advance to the next stage, heat stroke, which can be fatal. Heat exhaustion must be vigorously treated by Hypothermia is caused by exposure to cold. The urgency of having the victim lie down in a cool place, taking off as many treatment depends on the length of exposure and the victim's clothes as possible, cooling the victim with a cool water body temperature. Begin rewarming a victim of hypothermia spray, and encouraging the victim to drink cool fluids, immediately by moving the victim to a warm environment, preferably containing carbohydrates and electrolytes.
removing wet clothing, and wrapping all exposed body Heat stroke includes all the symptoms of heat exhaustion surfaces with anything at hand, such as blankets, clothing, plus signs of central nervous system involvement, including and newspapers. If the hypothermia victim is far from dizziness, syncope, confusion, or seizures. The most impor- definitive health care, begin active rewarming (Class IIa, tant action by a first aid provider for a victim of heat stroke LOE B159,160) although the effectiveness of active rewarming is to begin immediate cooling, preferably by immersing the has not been evaluated. Active rewarming should not delay victim up to the chin in cold water.186–189 It is also important definitive care. Potential methods of active rewarming in- to activate the EMS system. Heat stroke requires emergency November 2, 2010
treatment with intravenous fluids. Do not try to force the victim to drink liquids.
Brush powdered chemicals off the skin with a gloved hand orpiece of cloth. Remove all contaminated clothing from the victim, making sure you do not contaminate yourself in the Drowning is a major cause of unintentional death. Methods of process. In case of exposure to an acid or alkali on the skin196–202 preventing drowning include isolation fencing around swim- or eye,203–208 immediately irrigate the affected area with copious ming pools (gates should be self-closing and self-latching),190 amounts of water (Class I, LOE B).
wearing personal flotation devices (life jackets) while in,around, or on water, never swimming alone, and avoidingswimming or operating motorized watercraft while intoxi- Toxic Eye Injury
cated. Outcome following drowning depends on the duration Rinse eyes exposed to toxic substances immediately with a of the submersion, the water temperature, and how promptly copious amount of water (Class I, LOE C203,209,210), unless a CPR is started.191,192 Occasional case reports have docu- specific antidote is available.203,210,211 mented intact neurological survival in children followingprolonged submersion in icy waters.193,194 Remove the victim rapidly and safely from the water, but do not place yourself in danger. If you have special training, Treatment With Milk or Water
you can start rescue breathing while the victim is still in the Do not administer anything by mouth for any poison water195 providing that it does not delay removing the victim ingestion unless advised to do so by a poison control center from the water. There is no evidence that water acts as an or emergency medical personnel because it may be harmful obstructive foreign body, so do not waste time trying to (Class III, LOE C). There is insufficient evidence that remove it with abdominal or chest thrusts. Start CPR and, if dilution of the ingested poison with water or milk is of any you are alone, continue with about 5 cycles (about 2 minutes) benefit as a first aid measure. Animal studies212–216 have of chest compressions and ventilations before activating shown that dilution or neutralization of a caustic agent EMS. If 2 rescuers are present, send 1 rescuer to activateEMS immediately.
with water or milk reduces tissue injury, but no humanstudies have shown a clinical benefit. Possible adverseeffects of water or milk administration include emesis and If the patient exhibits any signs or symptoms of a life-threatening condition, (eg, sleepiness, seizures, difficulty breathing, vomiting) after exposure to a poison, activate the Do not administer activated charcoal to a victim who has EMS immediately.
ingested a poisonous substance unless you are advised to doso by poison control center or emergency medical personnel Poison Control Centers
(Class IIb, LOE C). There is no evidence that activated There are many poisonous substances in the home and charcoal is effective as a component of first aid. It may be worksite. It is important to understand the toxic nature of the safe to administer,217,218 but it has not been shown to be chemical substances in the environment and the proper beneficial, and there are reports of it causing harm.219–221 In protective equipment and emergency procedures in case of addition the majority of children will not take the recom- toxic exposure. The Poison Help hotline of the American Association of Poison Control Centers (800-222-1222) is anexcellent resource in the United States for information abouttreating ingestion of, or exposure to, a potential poison.
Further information is available at www.aapcc.org. Similar Do not administer syrup of ipecac for ingestions of toxins resources may be available internationally, and their contact (Class III, LOE B). Several studies223–225 found that there is information (eg, 112 in Europe) should be standard in no clinically relevant advantage to administering syrup of international first aid training. When phoning a poison control ipecac; its administration is not associated with decreased center or other emergency medical services, know the nature healthcare utilization.226 Untoward effects of ipecac admin- and time of exposure and the name of the product or toxic istration include intractable emesis and delayed care in an advanced medical facility.227,228 Markenson et al
Part 17: First Aid
Guidelines Part 17: First Aid: Writing Group Disclosures
NYMC–Interim Chair; EMA–Chief Brody School of Medicine, East *Serving as an expert witness in two ongoing lawsuits involving EMS related cases. Billing for this service has not yet occurred and will likely represent less than $10,000 per 12 months.
This payment is expected to come directly to me Emeritus Director Pediatric Cardiology, Connecticut Children's Medical Center, Clinical Professor, University of Connecticut French Red Cross, National Medical Advisor and Coordinator of the European Reference Centre for first aid education Coordinator of the Scientific Commission of First Aid for the French Interior Ministry Hong Kong Hospital Authority Hospital Chief Executive NorthEast Emergency Medical *Volunteer: American Red Services, Inc.– Regional EMS Cross Advisory Council Council: Provide EMS System on First Aid, Aquatics, Oversight. Also provide education Safety and Preparedness (First aid and CPR/AED) as an AHA (ACFASP) Vice-Chair Training Center. Executive Director; Isis Maternity: Pre-Natal and Post Partum edu.- Provide CPR and First Aid Training via AHA Curriculum.
City of Austin - Office of the Medical Director: EMS System Medical Director Staff–Performance Management & Research *Beginning July 1, 2009, I will serve as a paid consultant to the AHA ECC Product Development Group as a Senior Science Editor.
This assignment will include providing Science review of AHA First Aid Products Minute Clinic–Family Nurse Kent State University–Assistant Dir †Wilderness First Aid Faculty Professional Dev. Center consultant for StayWell Bloomington Hospital Prompt Stony Brook University-Physician This table represents the relationships of writing group members that may be perceived as actual or reasonably perceived conflicts of interest as reported on the Disclosure Questionnaire, which all members of the writing group are required to complete and submit. A relationship is considered to be "significant" if (a) the personreceives $10 000 or more during any 12-month period, or 5% or more of the person's gross income; or (b) the person owns 5% or more of the voting stock or shareof the entity, or owns $10 000 or more of the fair market value of the entity. A relationship is considered to be "modest" if it is less than "significant" under thepreceding definition.
November 2, 2010
26. Simon HK. Caregiver knowledge and delivery of a commonly pre- scribed medication (albuterol) for children. Arch Pediatr Adolesc Med.
1. 2005 American Heart Association guidelines for cardiopulmonary resuscitation and emergency cardiovascular care part 14: first aid.
27. Kim JS, Sinacore JM, Pongracic JA. Parental use of EpiPen for children 2. Markenson D, Ferguson JD, Chameides L, Cassan P, Chung KL, Epstein with food allergies. J Allergy Clin Immunol. 2005;116:164 –168.
JL, Gonzales L, Hazinski MF, Herrington RA, Pelligrino JL, Ratcliff N, 28. Sicherer SH, Simons FE. Quandaries in prescribing an emergency action Singer AJ; on behalf of the First Aid Chapter Collaborators. Part 13: first plan and self-injectable epinephrine for first-aid management of ana- aid: 2010 American Heart Association and American Red Cross Inter- phylaxis in the community. J Allergy Clin Immunol. 2005;115:575–583.
national Consensus on First Aid Science With Treatment Recommen- 29. Pouessel G, Deschildre A, Castelain C, Sardet A, Sagot-Bevenot S, de dations. Circulation. 2010;122(suppl 2):S582–S605.
Sauve-Boeuf A, Thumerelle C, Santos C. Parental knowledge and use of 3. Lewis TH. The Medicine Men: Oglala Sioux Ceremony and Healing. epinephrine auto-injector for children with food allergy. Pediatr Allergy Lincoln, Neb: University of Nebraska Press; 1992.
4. Pearn J. The earliest days of first aid. BMJ. 1994;309:1718 –1720.
30. Rainbow J, Browne GJ. Fatal asthma or anaphylaxis? Emerg Med J.
5. The American Heart Association in collaboration with the International 2002;19:415– 417.
Liaison Committee on Resuscitation. Guidelines 2000 for cardiopulmo- 31. Dobbie A, Robertson CM. Provision of self-injectable adrenaline for nary resuscitation and emergency cardiovascular care, part 5: new children at risk of anaphylaxis: its source, frequency and appropriateness guidelines for first aid. Circulation. 2000;102(suppl):I-77–I-85.
of use, and effect. Ambul Child Health. 1998;4:283–288.
6. Neely KW, Drake ME, Moorhead JC, Schmidt TA, Skeen DT, Wilson 32. Clegg SK, Ritchie JM. "Epipen" training: a survey of the provision for EA. Multiple options and unique pathways: a new direction for EMS? parents and teachers in West Lothian. Ambul Child Health. 2001;7: Ann Emerg Med. 1997;30:797–799.
7. Callaham M. Quantifying the scanty science of prehospital emergency 33. Gold MS, Sainsbury R. First aid anaphylaxis management in children care. Ann Emerg Med. 1997;30:785–790.
who were prescribed an epinephrine autoinjector device (EpiPen).
8. Spaite DW, Criss EA, Valenzuela TD, Meislin HW. Developing a J Allergy Clin Immunol. 2000;106(part 1):171–176.
foundation for the evaluation of expanded-scope EMS: a window of 34. Sicherer SH, Forman JA, Noone SA. Use assessment of self- opportunity that cannot be ignored. Ann Emerg Med. 1997;30:791–796.
administered epinephrine among food-allergic children and pedia- 9. Blake WE, Stillman BC, Eizenberg N, Briggs C, McMeeken JM. The tricians. Pediatrics. 2000;105:359 –362.
position of the spine in the recovery position: an experimental com- 35. Korenblat P, Lundie MJ, Dankner RE, Day JH. A retrospective study of parison between the lateral recovery position and the modified HAINES epinephrine administration for anaphylaxis: how many doses are position. Resuscitation. 2002;53:289 –297.
needed? Allergy Asthma Proc. 1999;20:383–386.
10. Gunn BD, Eizenberg N, Silberstein M, McMeeken JM, Tully EA, 36. Uguz A, Lack G, Pumphrey R, Ewan P, Warner J, Dick J, Briggs D, Stillman BC, Brown DJ, Gutteridge GA. How should an unconscious Clarke S, Reading D, Hourihane J. Allergic reactions in the community: person with a suspected neck injury be positioned? Prehosp Disaster a questionnaire survey of members of the anaphylaxis campaign. Clin Med. 1995;10:239 –244.
Exp Allergy. 2005;35:746 –750.
11. Wong DH, O'Connor D, Tremper KK, Zaccari J, Thompson P, Hill D.
37. Rudders SA, Banerji A, Corel B, Clark S, Camargo CA Jr. Multicenter Changes in cardiac output after acute blood loss and position change in study of repeat epinephrine treatments for food-related anaphylaxis.
man. Crit Care Med. 1989;17:979 –983.
Pediatrics. 2010;125:e711– e718.
12. Boulain T, Achard JM, Teboul JL, Richard C, Perrotin D, Ginies G.
38. Sicherer SH, Simons FE. Self-injectable epinephrine for first-aid man- Changes in BP induced by passive leg raising predict response to fluid agement of anaphylaxis. Pediatrics. 2007;119:638 – 646.
loading in critically ill patients. Chest. 2002;121:1245–1252.
39. Gaca AM, Frush DP, Hohenhaus SM, Luo X, Ancarana A, Pickles A, 13. Teboul JL, Monnet X. Prediction of volume responsiveness in critically Frush KS. Enhancing pediatric safety: using simulation to assess ill patients with spontaneous breathing activity. Curr Opin Crit Care.
radiology resident preparedness for anaphylaxis from intravenous 2008;14:334 –339.
contrast media. Radiology. 2007;245:236 –244.
14. Gaffney FA, Bastian BC, Thal ER, Atkins JM, Blomqvist CG. Passive 40. Pumphrey RS. Lessons for management of anaphylaxis from a study of leg raising does not produce a significant or sustained autotransfusion fatal reactions. Clin Exp Allergy. 2000;30:1144 –1150.
effect. J Trauma. 1982;22:190 –193.
41. Horowitz BZ, Jadallah S, Derlet RW. Fatal intracranial bleeding asso- 15. Ostrow CL. Use of the Trendelenburg position by critical care nurses: ciated with prehospital use of epinephrine. Ann Emerg Med. 1996;28: Trendelenburg survey. Am J Crit Care. 1997;6:172–176.
16. Shammas A, Clark AP. Trendelenburg positioning to treat acute hypo- 42. Davis CO, Wax PM. Prehospital epinephrine overdose in a child tension: helpful or harmful? Clin Nurse Spec. 2007;21:181–187.
resulting in ventricular dysrhythmias and myocardial ischemia. Pediatr 17. Reich DL, Konstadt SN, Raissi S, Hubbard M, Thys DM. Trendelenburg Emerg Care. 1999;15:116 –118.
position and passive leg raising do not significantly improve cardiopul- 43. Anchor J, Settipane RA. Appropriate use of epinephrine in anaphylaxis.
monary performance in the anesthetized patient with coronary artery Am J Emerg Med. 2004;22:488 – 490.
disease. Crit Care Med. 1989;17:313–317.
44. Zijlstra F, Ernst N, De Boer M-J, Nibbering E, Suryapranata H, Hoorntje 18. Johnson BA. Stark II, phase II: positive changes and lingering uncer- JCA, Dambrink J-HE, Van't Hof AWJ, Verheugt FWA. Influence of tainties. MGMA Connex. 2004;4:48 –51, 1.
prehospital administration of aspirin and heparin on initial patency of the 19. Rawles JM, Kenmure AC. Controlled trial of oxygen in uncomplicated infarct-related artery in patients with acute ST elevation myocardial myocardial infarction. BMJ. 1976;1:1121–1123.
infarction. J Am Coll Cardiol. 2002;39:1733–1737.
20. Nicholson C. A systematic review of the effectiveness of oxygen in 45. ISIS-2 (Second International Study of Infarct Survival) Collaborative reducing acute myocardial ischaemia. J Clin Nurs. 2004;13:996 –1007.
Group. Randomised trial of intravenous streptokinase, oral aspirin, both, 21. Austin M, Wood-Baker R. Oxygen therapy in the pre-hospital setting for or neither among 17,187 cases of suspected acute myocardial infarction: acute exacerbations of chronic obstructive pulmonary disease. Cochrane ISIS-2. Lancet. 1988;2:349 –360.
Database Syst Rev. 2006;3:CD005534.
46. Barbash IM, Freimark D, Gottlieb S, Hod H, Hasin Y, Battler A, Crystal 22. Longphre JM, Denoble PJ, Moon RE, Vann RD, Freiberger JJ. First aid E, Matetzky S, Boyko V, Mandelzweig L, Behar S, Leor J. Outcome of normobaric oxygen for the treatment of recreational diving injuries.
myocardial infarction in patients treated with aspirin is enhanced by Undersea Hyperb Med. 2007;34:43– 49.
pre-hospital administration. Cardiology. 2002;98:141–147.
23. Mannino DM, Homa DM, Pertowski CA, Ashizawa A, Nixon LL, 47. Lehmann KG, Heath-Lange SJ, Ferris ST. Randomized comparison of Johnson CA, Ball LB, Jack E, Kang DS. Surveillance for asthma: United hemostasis techniques after invasive cardiovascular procedures. Am States, 1960 –1995. MMWR CDC Surveill Summ. 1998;47:1–27.
Heart J. 1999;138(part 1):1118 –1125.
24. Hamid S, Kumaradevan J, Cochrane GM. Single centre open study to 48. Koreny M, Riedmuller E, Nikfardjam M, Siostrzonek P, Mullner M.
compare patient recording of PRN salbutamol use on a daily diary card Arterial puncture closing devices compared with standard manual com- with actual use as recorded by the MDI compliance monitor. Respir pression after cardiac catheterization: systematic review and meta-anal- Med. 1998;92:1188 –1190.
ysis. JAMA. 2004;291:350 –357.
25. O'Driscoll BR, Kay EA, Taylor RJ, Weatherby H, Chetty MC, Bernstein 49. Mlekusch W, Dick P, Haumer M, Sabeti S, Minar E, Schillinger M.
A. A long-term prospective assessment of home nebulizer treatment.
Arterial puncture site management after percutaneous transluminal pro- Respir Med. 1992;86:317–325.
cedures using a hemostatic wound dressing (Clo-Sur P.A.D.) versus Markenson et al
Part 17: First Aid
conventional manual compression: a randomized controlled trial.
73. Ersoy G, Kaynak MF, Yilmaz O, Rodoplu U, Maltepe F, Gokmen N.
J Endovasc Ther. 2006;13:23–31.
Hemostatic effects of microporous polysaccharide hemosphere in a rat 50. Upponi SS, Ganeshan AG, Warakaulle DR, Phillips-Hughes J, model with severe femoral artery bleeding. Adv Ther. 2007;24:485– 492.
Boardman P, Uberoi R. Angioseal versus manual compression for hae- 74. McManus J, Hurtado T, Pusateri A, Knoop KJ. A case series describing mostasis following peripheral vascular diagnostic and interventional thermal injury resulting from zeolite use for hemorrhage control in procedures: a randomized controlled trial. Eur J Radiol. 2007;61: combat operations. Prehosp Emerg Care. 2007;11:67–71.
75. Rhee P, Brown C, Martin M, Salim A, Plurad D, Green D, Chambers L, 51. Simon A, Bumgarner B, Clark K, Israel S. Manual versus mechanical Demetriades D, Velmahos G, Alam H. QuikClot use in trauma for compression for femoral artery hemostasis after cardiac catheterization.
hemorrhage control: case series of 103 documented uses. J Trauma.
Am J Crit Care. 1998;7:308 –313.
52. Walker SB, Cleary S, Higgins M. Comparison of the FemoStop device 76. Wedmore I, McManus JG, Pusateri AE, Holcomb JB. A special report and manual pressure in reducing groin puncture site complications on the chitosan-based hemostatic dressing: experience in current combat following coronary angioplasty and coronary stent placement. Int J Nurs operations. J Trauma. 2006;60:655– 658.
Pract. 2001;7:366 –375.
77. Dire DJ, Welsh AP. A comparison of wound irrigation solutions used in 53. Yadav JS, Ziada KM, Almany S, Davis TP, Castaneda F. Comparison of the emergency department. Ann Emerg Med. 1990;19:704 –708.
the QuickSeal Femoral Arterial Closure System with manual com- 78. Moscati R, Mayrose J, Fincher L, Jehle D. Comparison of normal saline pression following diagnostic and interventional catheterization pro- with tap water for wound irrigation. Am J Emerg Med. 1998;16: cedures. Am J Cardiol. 2003;91:1463–1466, A1466.
54. Naimer SA, Chemla F. Elastic adhesive dressing treatment of bleeding 79. Bansal BC, Wiebe RA, Perkins SD, Abramo TJ. Tap water for irrigation wounds in trauma victims. Am J Emerg Med. 2000;18:816 – 819.
of lacerations. Am J Emerg Med. 2002;20:469 – 472.
55. Pillgram-Larsen J, Mellesmo S. Not a tourniquet, but compressive dress- 80. Valente JH, Forti RJ, Freundlich LF, Zandieh SO, Crain EF. Wound ing: experience from 68 traumatic amputations after injuries from mines irrigation in children: saline solution or tap water? Ann Emerg Med.
[in Norwegian]. Tidsskr Nor Laegeforen. 1992;112:2188 –2190.
2003;41:609 – 616.
56. Naimer SA, Nash M, Niv A, Lapid O. Control of massive bleeding from 81. Moscati RM, Mayrose J, Reardon RF, Janicke DM, Jehle DV. A mul- facial gunshot wound with a compact elastic adhesive compression ticenter comparison of tap water versus sterile saline for wound irri- dressing. Am J Emerg Med. 2004;22:586 –588.
gation. Acad Emerg Med. 2007;14:404 – 409.
57. Naimer SA, Anat N, Katif G. Evaluation of techniques for treating the 82. Longmire AW, Broom LA, Burch J. Wound infection following high- bleeding wound. Injury. 2004;35:974 –979.
pressure syringe and needle irrigation. Am J Emerg Med. 1987;5: 58. Lakstein D, Blumenfeld A, Sokolov T, Lin G, Bssorai R, Lynn M, Ben-Abraham R. Tourniquets for hemorrhage control on the battlefield: 83. Claus EE, Fusco CF, Ingram T, Ingersoll CD, Edwards JE, Melham a 4-year accumulated experience. J Trauma. 2003;54(suppl): TJ. Comparison of the effects of selected dressings on the healing of standardized abrasions. J Athl Train. 1998;33:145–149.
59. Beekley AC, Sebesta JA, Blackbourne LH, Herbert GS, Kauvar DS, 84. Beam JW. Occlusive dressings and the healing of standardized Baer DG, Walters TJ, Mullenix PS, Holcomb JB. Prehospital tourniquet abrasions. J Athl Train. 2008;43:600 – 607.
use in Operation Iraqi Freedom: effect on hemorrhage control and 85. Hinman CD, Maibach H. Effect of air exposure and occlusion on outcomes. J Trauma. 2008;64(suppl):S28 –S37.
experimental human skin wounds. Nature. 1963;200:377–378.
60. Kragh JF Jr, Walters TJ, Baer DG, Fox CJ, Wade CE, Salinas J, 86. Huang HM, Wang JH, Yang L, Yi ZH. Effect of local treatment with Holcomb JB. Practical use of emergency tourniquets to stop bleeding in cooling and spray film on early edema of superficial II degree scald major limb trauma. J Trauma. 2008;64(suppl):S38 –S49.
burns in rats [in Chinese]. Nan Fang Yi Ke Da Xue Xue Bao. 2009;29: 61. Kalish J, Burke P, Feldman J, Agarwal S, Glantz A, Moyer P, Serino R, 804 – 806.
Hirsch E. The return of tourniquets: original research evaluates the 87. Cuttle L, Kempf M, Kravchuk O, Phillips GE, Mill J, Wang XQ, Kimble effectiveness of prehospital tourniquets for civilian penetrating RM. The optimal temperature of first aid treatment for partial thickness extremity injuries. JEMS. 2008;33:44 – 46, 49 –50, 52, 54.
burn injuries. Wound Repair Regen. 2008;16:626 – 634.
62. Savvidis E, Parsch K. Prolonged transitory paralysis after pneumatic 88. Yuan J, Wu C, Holland AJ, Harvey JG, Martin HC, La Hei ER, tourniquet use on the upper arm [in German]. Unfallchirurg. 1999;102: Arbuckle S, Godfrey TC. Assessment of cooling on an acute scald burn injury in a porcine model. J Burn Care Res. 2007;28:514 –520.
63. Kornbluth ID, Freedman MK, Sher L, Frederick RW. Femoral, 89. Ofeigsson OJ, Mitchell R, Patrick RS. Observations on the cold water saphenous nerve palsy after tourniquet use: a case report. Arch Phys Med treatment of cutaneous burns. J Pathol. 1972;108:145–150.
Rehabil. 2003;84:909 –911.
90. Nguyen NL, Gun RT, Sparnon AL, Ryan P. The importance of 64. Landi A, Saracino A, Pinelli M, Caserta G, Facchini MC. Tourniquet immediate cooling: a case series of childhood burns in Vietnam. Burns.
paralysis in microsurgery. Ann Acad Med Singapore. 1995;24(suppl): 91. Tung KY, Chen ML, Wang HJ, Chen GS, Peck M, Yang J, Liu CC. A 65. Wakai A, Wang JH, Winter DC, Street JT, O'Sullivan RG, Redmond seven-year epidemiology study of 12,381 admitted burn patients in HP. Tourniquet-induced systemic inflammatory response in extremity Taiwan: using the Internet registration system of the Childhood Burn surgery. J Trauma. 2001;51:922–926.
Foundation. Burns. 2005;31(suppl 1):S12–S17.
66. Mohler LR, Pedowitz RA, Lopez MA, Gershuni DH. Effects of tour- 92. Li C, Yu D, Li MS. Clinical and experiment study of cooling therapy on niquet compression on neuromuscular function. Clin Orthop Relat Res. burned wound [in Chinese]. Zhonghua Yi Xue Za Zhi. 1997;77:586 –588.
Feb 1999:213–220.
93. Matthews RN, Radakrishnan T. First-aid for burns. Lancet. 1987; 67. Kokki H, Vaatainen U, Penttila I. Metabolic effects of a low-pressure tourniquet system compared with a high-pressure tourniquet system in 94. Purdue GF, Layton TR, Copeland CE. Cold injury complicating burn arthroscopic anterior crucial ligament reconstruction. Acta Anaesthesiol therapy. J Trauma. 1985;25:167–168.
Scand. 1998;42:418 – 424.
95. Sawada Y, Urushidate S, Yotsuyanagi T, Ishita K. Is prolonged and 68. King RB, Filips D, Blitz S, Logsetty S. Evaluation of possible tourniquet excessive cooling of a scalded wound effective? Burns. 1997;23:55–58.
systems for use in the Canadian Forces. J Trauma. 2006;60:1061–1071.
96. Swain AH, Azadian BS, Wakeley CJ, Shakespeare PG. Management of 69. Wenke JC, Walters TJ, Greydanus DJ, Pusateri AE, Convertino VA.
blisters in minor burns. BMJ (Clin Res Ed). 1987;295:181.
Physiological evaluation of the U.S. Army one-handed tourniquet. Mil 97. Cope O. The treatment of the surface burns. Ann Surg. 1943;117: Med. 2005;170:776 –781.
70. Calkins D, Snow C, Costello M, Bentley TB. Evaluation of possible 98. Forage AV. The effects of removing the epidermis from burnt skin.
battlefield tourniquet systems for the far-forward setting. Mil Med.
Lancet. 1962;2:690 – 693.
99. Gimbel NS, Kapetansky DI, Weissman F, Pinkus HK. A study of 71. Walters TJ, Wenke JC, Kauvar DS, McManus JG, Holcomb JB, Baer epithelization in blistered burns. AMA Arch Surg. 1957;74:800 – 803.
DG. Effectiveness of self-applied tourniquets in human volunteers.
100. Homma S, Gillam LD, Weyman AE. Echocardiographic observations in Prehosp Emerg Care. 2005;9:416 – 422.
survivors of acute electrical injury. Chest. 1990;97:103–105.
72. Swan KG Jr, Wright DS, Barbagiovanni SS, Swan BC, Swan KG.
101. Jensen PJ, Thomsen PE, Bagger JP, Norgaard A, Baandrup U. Electrical Tourniquets revisited. J Trauma. 2009;66:672– 675.
injury causing ventricular arrhythmias. Br Heart J. 1987;57:279 –283.
November 2, 2010
102. Lowery DW, Wald MM, Browne BJ, Tigges S, Hoffman JR, Mower 124. McMaster WC, Liddle S, Waugh TR. Laboratory evaluation of various WR. Epidemiology of cervical spine injury victims. Ann Emerg Med.
cold therapy modalities. Am J Sports Med. 1978;6:291–294.
125. Chesterton LS, Foster NE, Ross L. Skin temperature response to cryo- 103. Stiell IG, Wells GA, Vandemheen KL, Clement CM, Lesiuk H, De Maio therapy. Arch Phys Med Rehabil. 2002;83:543–549.
VJ, Laupacis A, Schull M, McKnight RD, Verbeek R, Brison R, Cass D, 126. Graham CA, Stevenson J. Frozen chips: an unusual cause of severe Dreyer J, Eisenhauer MA, Greenberg GH, MacPhail I, Morrison L, frostbite injury. Br J Sports Med. 2000;34:382–383.
Reardon M, Worthington J. The Canadian C-spine rule for radiography 127. Moeller JL, Monroe J, McKeag DB. Cryotherapy-induced common in alert and stable trauma patients. JAMA. 2001;286:1841–1848.
peroneal nerve palsy. Clin J Sport Med. 1997;7:212–216.
104. Hackl W, Hausberger K, Sailer R, Ulmer H, Gassner R. Prevalence of 128. Bassett FH III, Kirkpatrick JS, Engelhardt DL, Malone TR.
cervical spine injuries in patients with facial trauma. Oral Surg Oral Cryotherapy-induced nerve injury. Am J Sports Med. 1992;20:516 –518.
Med Oral Pathol Oral Radiol Endod. 2001;92:370 –376.
129. Bleakley CM, McDonough SM, MacAuley DC, Bjordal J. Cryotherapy 105. Kennedy E. Spinal Cord Injury: The Facts and Figures. Birmingham, for acute ankle sprains: a randomised controlled study of two different Ala: University of Alabama Press; 1986.
icing protocols. Br J Sports Med. 2006;40:700 –705.
106. Reid DC, Henderson R, Saboe L, Miller JD. Etiology and clinical course 130. Auerbach PS, Geehr EC, Ryu RK. The Reel Splint: experience with a of missed spine fractures. J Trauma. 1987;27:980 –986.
new traction splint apparatus in the prehospital setting. Ann Emerg Med.
107. Davis JW, Phreaner DL, Hoyt DB, Mackersie RC. The etiology of 1984;13:419 – 422.
missed cervical spine injuries. J Trauma. 1993;34:342–346.
131. Kaplan MM, Cohen D, Koprowski H, Dean D, Ferrigan L. Studies on 108. Hauswald M, Ong G, Tandberg D, Omar Z. Out-of-hospital spinal the local treatment of wounds for the prevention of rabies. Bull World immobilization: its effect on neurologic injury. Acad Emerg Med. 1998; Health Organ. 1962;26:765–775.
5:214 –219.
132. Dean DJ, Baer GM, Thompson WR. Studies on the local treatment of 109. Hoffman JR, Mower WR, Wolfson AB, Todd KH, Zucker MI; National rabies-infected wounds. Bull World Health Organ. 1963;28:477– 486.
Emergency X-Radiography Utilization Study Group. Validity of a set of 133. Callaham ML. Treatment of common dog bites: infection risk factors.
clinical criteria to rule out injury to the cervical spine in patients with JACEP. 1978;7:83– 87.
blunt trauma. N Engl J Med. 2000;343:94 –99.
134. Alberts MB, Shalit M, LoGalbo F. Suction for venomous snakebite: a 110. Viccellio P, Simon H, Pressman BD, Shah MN, Mower WR, Hoffman study of "mock venom" extraction in a human model. Ann Emerg Med.
JR. A prospective multicenter study of cervical spine injury in children.
135. Lawrence WT, Giannopoulos A, Hansen A. Pit viper bites: rational 111. Touger M, Gennis P, Nathanson N, Lowery DW, Pollack CV Jr, management in locales in which copperheads and cottonmouths pre-dominate. Ann Plast Surg. 1996;36:276 –285.
Hoffman JR, Mower WR. Validity of a decision rule to reduce cervical 136. Leopold RS, Huber GS. Ineffectiveness of suction in removing snake spine radiography in elderly patients with blunt trauma. Ann Emerg venom from open wounds. U S Armed Forces Med J. 1960;11:682– 685.
137. Bush SP, Hegewald KG, Green SM, Cardwell MD, Hayes WK. Effects 112. Panacek EA, Mower WR, Holmes JF, Hoffman JR. Test performance of of a negative pressure venom extraction device (Extractor) on local the individual NEXUS low-risk clinical screening criteria for cervical tissue injury after artificial rattlesnake envenomation in a porcine model.
spine injury. Ann Emerg Med. 2001;38:22–25.
Wilderness Environ Med. 2000;11:180 –188.
113. Pieretti-Vanmarcke R, Velmahos GC, Nance ML, Islam S, Falcone RA 138. Holstege CP, Singletary EM. Images in emergency medicine: skin Jr, Wales PW, Brown RL, Gaines BA, McKenna C, Moore FO, Goslar damage following application of suction device for snakebite. Ann PW, Inaba K, Barmparas G, Scaife ER, Metzger RR, Brockmeyer DL, Emerg Med. 2006;48:105, 113.
Upperman JS, Estrada J, Lanning DA, Rasmussen SK, Danielson PD, 139. Howarth DM, Southee AE, Whyte IM. Lymphatic flow rates and Hirsh MP, Consani HF, Stylianos S, Pineda C, Norwood SH, Bruch SW, first-aid in simulated peripheral snake or spider envenomation. Med J Drongowski R, Barraco RD, Pasquale MD, Hussain F, Hirsch EF, McNeely PD, Fallat ME, Foley DS, Iocono JA, Bennett HM, Waxman 140. German BT, Hack JB, Brewer K, Meggs WJ. Pressure-immobilization K, Kam K, Bakhos L, Petrovick L, Chang Y, Masiakos PT. Clinical bandages delay toxicity in a porcine model of eastern coral snake clearance of the cervical spine in blunt trauma patients younger than 3 (Micrurus fulvius fulvius) envenomation. Ann Emerg Med. 2005;45: years: a multi-center study of the American Association for the Surgery of Trauma. J Trauma. 2009;67:543–549.
141. Bush SP, Green SM, Laack TA, Hayes WK, Cardwell MD, Tanen DA.
114. Domeier RM, Evans RW, Swor RA, Hancock JB, Fales W, Krohmer J, Pressure immobilization delays mortality and increases intracompart- Frederiksen SM, Shork MA. The reliability of prehospital clinical eval- mental pressure after artificial intramuscular rattlesnake envenomation uation for potential spinal injury is not affected by the mechanism of in a porcine model. Ann Emerg Med. 2004;44:599 – 604.
injury. Prehosp Emerg Care. 1999;3:332–337.
142. Norris RL, Ngo J, Nolan K, Hooker G. Physicians and lay people are 115. Cote DJ, Prentice WE Jr, Hooker DN, Shields EW. Comparison of three unable to apply pressure immobilization properly in a simulated treatment procedures for minimizing ankle sprain swelling. Phys Ther.
snakebite scenario. Wilderness Environ Med. 2005;16:16 –21.
143. Simpson ID, Tanwar PD, Andrade C, Kochar DK, Norris RL. The 116. Meeusen R, Lievens P. The use of cryotherapy in sports injuries. Sports Ebbinghaus retention curve: training does not increase the ability to Med. 1986;3:398 – 414.
apply pressure immobilisation in simulated snake bite: implications for 117. Hocutt JE Jr, Jaffe R, Rylander CR, Beebe JK. Cryotherapy in ankle snake bite first aid in the developing world. Trans R Soc Trop Med Hyg.
sprains. Am J Sports Med. 1982;10:316 –319.
118. Airaksinen OV, Kyrklund N, Latvala K, Kouri JP, Gronblad M, Kolari 144. Mianzan HW, Fenner PJ, Cornelius PF, Ramirez FC. Vinegar as a P. Efficacy of cold gel for soft tissue injuries: a prospective randomized disarming agent to prevent further discharge of the nematocysts of the double-blinded trial. Am J Sports Med. 2003;31:680 – 684.
stinging hydromedusa Olindias sambaquiensis. Cutis. 2001;68:45– 48.
119. Basur RL, Shephard E, Mouzas GL. A cooling method in the treatment 145. Burnett JW, Rubinstein H, Calton GJ. First aid for jellyfish enveno- of ankle sprains. Practitioner. 1976;216:708 –711.
mation. South Med J. 1983;76:870 – 872.
120. Ayata R, Shiraki H, Fukuda T, Takemura M, Mukai N, Miyakawa S.
146. Loten C, Stokes B, Worsley D, Seymour JE, Jiang S, Isbister GK. A The effects of icing after exercise on jumper's knee. Jpn J Phys Fitness randomised controlled trial of hot water (45 degrees C) immersion Sports Med. 2007;56:125–130.
versus ice packs for pain relief in bluebottle stings. Med J Aust. 2006; 121. Merrick MA, Jutte LS, Smith ME. Cold modalities with different ther- 184:329 –333.
modynamic properties produce different surface and intramuscular tem- 147. Nomura JT, Sato RL, Ahern RM, Snow JL, Kuwaye TT, Yamamoto LG.
peratures. J Athl Train. 2003;38:28 –33.
A randomized paired comparison trial of cutaneous treatments for acute 122. Dykstra JH, Hill HM, Miller MG, Cheatham CC, Michael TJ, Baker jellyfish (Carybdea alata) stings. Am J Emerg Med. 2002;20:624 – 626.
RJ. Comparisons of cubed ice, crushed ice, and wetted ice on intra- 148. Yoshimoto CM, Yanagihara AA. Cnidarian (coelenterate) enveno- muscular and surface temperature changes. J Athl Train. 2009;44: mations in Hawai'i improve following heat application. Trans R Soc Trop Med Hyg. 2002;96:300 –303.
123. Kanlayanaphotporn R, Janwantanakul P. Comparison of skin surface 149. Atkinson PR, Boyle A, Hartin D, McAuley D. Is hot water immersion an temperature during the application of various cryotherapy modalities.
effective treatment for marine envenomation? Emerg Med J. 2006;23: Arch Phys Med Rehabil. 2005;86:1411–1415.
Markenson et al
Part 17: First Aid
150. Thomas J. Dermatology in the new millennium. Indian J Dermatol 175. Foray J. Mountain frostbite: current trends in prognosis and treatment Venereol Leprol. 2001;67:100 –103.
(from results concerning 1261 cases). Int J Sports Med. 1992;13(suppl 151. Exton DR, Fenner PJ, Williamson JA. Cold packs: effective topical analgesia in the treatment of painful stings by Physalia and other jel- 176. Kenefick RW, O'Moore KM, Mahood NV, Castellani JW. Rapid IV lyfish. Med J Aust. 1989;151:625– 626.
versus oral rehydration: responses to subsequent exercise heat stress.
152. Thomas CS, Scott SA, Galanis DJ, Goto RS. Box jellyfish (Carybdea Med Sci Sports Exerc. 2006;38:2125–2131.
alata) in Waikiki: the analgesic effect of sting-aid, Adolph's meat ten- 177. Michell MW, Oliveira HM, Kinsky MP, Vaid SU, Herndon DN, Kramer derizer and fresh water on their stings: a double-blinded, randomized, GC. Enteral resuscitation of burn shock using World Health Organi- placebo-controlled clinical trial. Hawaii Med J. 2001;60:205–207, 210.
zation oral rehydration solution: a potential solution for mass casualty 153. Pereira PL, Carrette T, Cullen P, Mulcahy RF, Little M, Seymour J.
care. J Burn Care Res. 2006;27:819 – 825.
Pressure immobilisation bandages in first-aid treatment of jellyfish 178. Barclay RL, Depew WT, Vanner SJ. Carbohydrate-electrolyte rehy- envenomation: current recommendations reconsidered. Med J Aust.
dration protects against intravascular volume contraction during colonic 2000;173:650 – 652.
cleansing with orally administered sodium phosphate. Gastrointest 154. Seymour J, Carrette T, Cullen P, Little M, Mulcahy RF, Pereira PL. The Endosc. 2002;56:633– 638.
use of pressure immobilization bandages in the first aid management of 179. Currell K, Urch J, Cerri E, Jentjens RL, Blannin AK, Jeukendrup AE.
cubozoan envenomings. Toxicon. 2002;40:1503–1505.
Plasma deuterium oxide accumulation following ingestion of different 155. Flores MT. Traumatic injuries in the primary dentition. Dent Traumatol.
carbohydrate beverages. Appl Physiol Nutr Metab. 2008;33:1067–1072.
180. Jeukendrup AE, Currell K, Clarke J, Cole J, Blannin AK. Effect of 156. Hiltz J, Trope M. Vitality of human lip fibroblasts in milk, Hanks beverage glucose and sodium content on fluid delivery. Nutr Metab balanced salt solution and Viaspan storage media. Endod Dent Traumatol. 1991;7:69 –72.
181. Evans GH, Shirreffs SM, Maughan RJ. Postexercise rehydration in man: 157. Chan AW, Wong TK, Cheung GS. Lay knowledge of physical education the effects of osmolality and carbohydrate content of ingested drinks.
teachers about the emergency management of dental trauma in Hong Kong. Dent Traumatol. 2001;17:77– 85.
182. Greenleaf JE, Jackson CG, Geelen G, Keil LC, Hinghofer-Szalkay H, 158. Sae-Lim V, Lim LP. Dental trauma management awareness of Sin- Whittam JH. Plasma volume expansion with oral fluids in hypohydrated gapore pre-school teachers. Dent Traumatol. 2001;17:71–76.
men at rest and during exercise. Aviat Space Environ Med. 1998;69: 159. Greif R, Rajek A, Laciny S, Bastanmehr H, Sessler DI. Resistive heating is more effective than metallic-foil insulation in an experimental model 183. Maughan RJ, Leiper JB. Sodium intake and post-exercise rehydration in of accidental hypothermia: a randomized controlled trial. Ann Emerg man. Eur J Appl Physiol Occup Physiol. 1995;71:311–319.
184. Merson SJ, Maughan RJ, Shirreffs SM. Rehydration with drinks dif- 160. Steele MT, Nelson MJ, Sessler DI, Fraker L, Bunney B, Watson WA, fering in sodium concentration and recovery from moderate exercise- Robinson WA. Forced air speeds rewarming in accidental hypothermia.
induced hypohydration in man. Eur J Appl Physiol. 2008;103:585–594.
Ann Emerg Med. 1996;27:479 – 484.
185. Shirreffs SM, Taylor AJ, Leiper JB, Maughan RJ. Post-exercise rehy- 161. Mills WJ Jr, Whaley R, Fish W. Frostbite: experience with rapid dration in man: effects of volume consumed and drink sodium content.
rewarming and ultrasonic therapy: part II: 1960. Alaska Med. 1993;35: Med Sci Sports Exerc. 1996;28:1260 –1271.
186. Clapp AJ, Bishop PA, Muir I, Walker JL. Rapid cooling techniques in 162. Mills WJ Jr, Whaley R, Fish W. Frostbite: experience with rapid joggers experiencing heat strain. J Sci Med Sport. 2001;4:160 –167.
rewarming and ultrasonic therapy: part III: 1961. Alaska Med. 1993;35: 187. Clements JM, Casa DJ, Knight J, McClung JM, Blake AS, Meenen PM, Gilmer AM, Caldwell KA. Ice-water immersion and cold-water 163. Fuhrman FA, Crismon JM. Studies on gangrene following cold injury: immersion provide similar cooling rates in runners with exercise- treatment of cold injury by means of immediate rapid warming. J Clin induced hyperthermia. J Athl Train. 2002;37:146 –150.
Invest. 1947;26:476 – 485.
188. Proulx CI, Ducharme MB, Kenny GP. Effect of water temperature on 164. Entin MA, Baxter H. Influence of rapid warming on frostbite in exper- cooling efficiency during hyperthermia in humans. J Appl Physiol.
imental animals. Plast Reconstr Surg (1946). 1952;9:511–524.
165. Fuhrman FA, Fuhrman GJ. The treatment of experimental frostbite by 189. Armstrong LE, Crago AE, Adams R, Roberts WO, Maresh CM.
rapid thawing: a review and new experimental data. Medicine (Bal- Whole-body cooling of hyperthermic runners: comparison of two field timore). 1957;36:465– 487.
166. Malhotra MS, Mathew L. Effect of rewarming at various water bath therapies. Am J Emerg Med. 1996;14:355–358.
temperatures in experimental frostbite. Aviat Space Environ Med. 1978; 190. American Academy of Pediatrics Committee on Injury, Violence, and 49:874 – 876.
Poison Prevention. Prevention of drowning in infants, children, and 167. Purkayastha SS, Chhabra PC, Verma SS, Selvamurthy W. Experimental adolescents. Pediatrics. 2003;112:437– 439.
studies on the treatment of frostbite in rats. Indian J Med Res. 1993;98: 191. Suominen P, Baillie C, Korpela R, Rautanen S, Ranta S, Olkkola KT.
Impact of age, submersion time and water temperature on outcome in 168. Martinez Villen G, Garcia Bescos G, Rodriguez Sosa V, Morandeira Garcia JR. Effects of haemodilution and rewarming with regard to 192. Graf WD, Cummings P, Quan L, Brutocao D. Predicting outcome in digital amputation in frostbite injury: an experimental study in the rabbit.
pediatric submersion victims. Ann Emerg Med. 1995;26:312–319.
J Hand Surg Br. 2002;27:224 –228.
193. Modell JH, Idris AH, Pineda JA, Silverstein JH. Survival after pro- 169. Purkayastha SS, Bhaumik G, Chauhan SK, Banerjee PK, Selvamurthy longed submersion in freshwater in Florida. Chest. 2004;125: W. Immediate treatment of frostbite using rapid rewarming in tea 1948 –1951.
decoction followed by combined therapy of pentoxifylline, aspirin & 194. Mehta SR, Srinivasan KV, Bindra MS, Kumar MR, Lahiri AK. Near vitamin C. Indian J Med Res. 2002;116:29 –34.
drowning in cold water. J Assoc Physicians India. 2000;48:674 – 676.
170. Bilgic S, Ozkan H, Ozenc S, Safaz I, Yildiz C. Treating frostbite. Can 195. Szpilman D, Soares M. In-water resuscitation: is it worthwhile? Fam Physician. 2008;54:361–363.
171. Sands WA, Kimmel WL, Wurtz BR, Stone MH, McNeal JR. Com- 196. Latenser BA, Lucktong TA. Anhydrous ammonia burns: case presenta- parison of commercially available disposable chemical hand and foot tion and literature review. J Burn Care Rehabil. 2000;21(part 1):40 – 42.
warmers. Wilderness Environ Med. 2009;20:33–38.
197. Wibbenmeyer LA, Morgan LJ, Robinson BK, Smith SK, Lewis RW II, 172. Heggers JP, Robson MC, Manavalen K, Weingarten MD, Carethers JM, Kealey GP. Our chemical burn experience: exposing the dangers of Boertman JA, Smith DJ Jr, Sachs RJ. Experimental and clinical obser- anhydrous ammonia. J Burn Care Rehabil. 1999;20:226 –231.
vations on frostbite. Ann Emerg Med. 1987;16:1056 –1062.
198. Yano K, Hosokawa K, Kakibuchi M, Hikasa H, Hata Y. Effects of 173. McCauley RL, Hing DN, Robson MC, Heggers JP. Frostbite injuries: a washing acid injuries to the skin with water: an experimental study using rational approach based on the pathophysiology. J Trauma. 1983;23: rats. Burns. 1995;21:500 –502.
199. Kono K, Yoshida Y, Watanabe M, Tanioka Y, Dote T, Orita Y, Bessho 174. Twomey JA, Peltier GL, Zera RT. An open-label study to evaluate the Y, Yoshida J, Sumi Y, Umebayashi K. An experimental study on the safety and efficacy of tissue plasminogen activator in treatment of severe treatment of hydrofluoric acid burns. Arch Environ Contam Toxicol.
frostbite. J Trauma. 2005;59:1350 –1354.
1992;22:414 – 418.
November 2, 2010
200. Murao M. Studies on the treatment of hydrofluoric acid burn. Bull 215. Homan CS, Singer AJ, Henry MC, Thode HC Jr. Thermal effects of Osaka Med Coll. 1989;35:39 – 48.
neutralization therapy and water dilution for acute alkali exposure in 201. Lorette JJ Jr, Wilkinson JA. Alkaline chemical burn to the face requiring canines. Acad Emerg Med. 1997;4:27–32.
full-thickness skin grafting. Ann Emerg Med. 1988;17:739 –741.
216. Homan CS, Singer AJ, Thomajan C, Henry MC, Thode HC Jr. Thermal 202. Leonard LG, Scheulen JJ, Munster AM. Chemical burns: effect of characteristics of neutralization therapy and water dilution for strong prompt first aid. J Trauma. 1982;22:420 – 423.
acid ingestion: an in-vivo canine model. Acad Emerg Med. 1998;5: 203. Kompa S, Schareck B, Tympner J, Wustemeyer H, Schrage NF. Com- parison of emergency eye-wash products in burned porcine eyes.
217. Spiller HA, Rodgers GC Jr. Evaluation of administration of activated Graefes Arch Clin Exp Ophthalmol. 2002;240:308 –313.
charcoal in the home. Pediatrics. 2001;108:E100.
204. McCulley JP. Ocular hydrofluoric acid burns: animal model, mechanism 218. Lamminpaa A, Vilska J, Hoppu K. Medical charcoal for a child's of injury and therapy. Trans Am Ophthalmol Soc. 1990;88:649 – 684.
poisoning at home: availability and success of administration in Finland.
205. Hojer J, Personne M, Hulten P, Ludwigs U. Topical treatments for Hum Exp Toxicol. 1993;12:29 –32.
hydrofluoric acid burns: a blind controlled experimental study. J Toxicol 219. Donoso A, Linares M, Leon J, Rojas G, Valverde C, Ramirez M, Clin Toxicol. 2002;40:861– 866.
Oberpaur B. Activated charcoal laryngitis in an intubated patient.
206. Herr RD, White GL Jr, Bernhisel K, Mamalis N, Swanson E. Clinical Pediatr Emerg Care. 2003;19:420 – 421.
comparison of ocular irrigation fluids following chemical injury. Am J 220. Dorrington CL, Johnson DW, Brant R. The frequency of complications Emerg Med. 1991;9:228 –231.
associated with the use of multiple-dose activated charcoal. Ann Emerg 207. Ingram TA III. Response of the human eye to accidental exposure to Med. 2003;41:370 –377.
sodium hypochlorite. J Endod. 1990;16:235–238.
221. Givens T, Holloway M, Wason S. Pulmonary aspiration of activated 208. Burns FR, Paterson CA. Prompt irrigation of chemical eye injuries may charcoal: a complication of its misuse in overdose management. Pediatr avert severe damage. Occup Health Saf. 1989;58:33–36.
Emerg Care. 1992;8:137–140.
209. Kompa S, Redbrake C, Hilgers C, Wustemeyer H, Schrage N, Remky A.
222. Scharman EJ, Cloonan HA, Durback-Morris LF. Home administration Effect of different irrigating solutions on aqueous humour pH changes, of charcoal: can mothers administer a therapeutic dose? J Emerg Med.
intraocular pressure and histological findings after induced alkali burns.
Acta Ophthalmol Scand. 2005;83:467– 470.
223. Kulig K, Bar-Or D, Cantrill SV, Rosen P, Rumack BH. Management of 210. Spoler F, Frentz M, Forst M, Kurz H, Schrage NF. Analysis of hydro- acutely poisoned patients without gastric emptying. Ann Emerg Med.
fluoric acid penetration and decontamination of the eye by means of time-resolved optical coherence tomography. Burns. 2008;34:549 –555.
224. Pond SM, Lewis-Driver DJ, Williams GM, Green AC, Stevenson NW.
211. Rihawi S, Frentz M, Schrage NF. Emergency treatment of eye burns: Gastric emptying in acute overdose: a prospective randomised con- which rinsing solution should we choose? Graefes Arch Clin Exp Oph- trolled trial. Med J Aust. 1995;163:345–349.
thalmol. 2006;244:845– 854.
225. Caravati EM. Unintentional acetaminophen ingestion in children and the 212. Homan CS, Maitra SR, Lane BP, Geller ER. Effective treatment of acute potential for hepatotoxicity. J Toxicol Clin Toxicol. 2000;38:291–296.
alkali injury of the rat esophagus with early saline dilution therapy. Ann 226. Bond G. Home syrup of ipecac use does not reduce emergency Emerg Med. 1993;22:178 –182.
department use or improve outcome. Pediatrics. 2003;112:1061–1064.
213. Homan CS, Maitra SR, Lane BP, Thode HC, Sable M. Therapeutic 227. Kornberg AE, Dolgin J. Pediatric ingestions: charcoal alone versus effects of water and milk for acute alkali injury of the esophagus. Ann ipecac and charcoal. Ann Emerg Med. 1991;20:648 – 651.
Emerg Med. 1994;24:14 –20.
228. Czajka PA, Russell SL. Nonemetic effects of ipecac syrup. Pediatrics.
214. Homan CS, Maitra SR, Lane BP, Thode HC Jr, Davidson L. Histo- pathologic evaluation of the therapeutic efficacy of water and milkdilution for esophageal acid injury. Acad Emerg Med. 1995;2:587–591.
KEY WORDS: emergency 䡲 injury

Source: http://hlr.nu/wp-content/uploads/AHA-2010-first-aid-guidelines-.pdf

Notas a los estados financieros a diciembre 31 de 1999

EMPRESA DE DESARROLLO URBANO DE BOLÍVAR S.A. NIT: 890.481.123-1 NOTAS A LOS ESTADOS FINANCIEROS A JUNIO 30 DE 2015 1. NOTAS DE CARÁCTER GENERAL 1. NATURALEZA JURIDICA, OBJETO SOCIAL ACTIVIDADES QUE DESAROLLA O COMETIDO ESTATAL. NATURALEZ-A JURIDICA La Empresa de Desarrollo Urbano de Bolívar -EDURBE S.A., es una Empresa Industrial y Comercial del estado, del Orden Distrital, constituida el 24 de Diciembre de 1981, mediante Escritura Número 2069 de la Notaria 2da de Cartagena, su capital es netamente público y sus

prodonto.com.ar

Full-mouth disinfection: another choice for periodontal therapy Artículo extraído de la revista RAOA, Revista de la Asociación Odontológica Argentina, Vol. 97 – Nº4 – Agosto/Septiembre de 2009 Resumen El raspaje radicular realizado por sectores ha mostrado mejorar el cuadro de la patología gingivo-periodontal. Este insume varias sesiones y determinado tiempo de tratamiento.